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- W2904641366 endingPage "228" @default.
- W2904641366 startingPage "183" @default.
- W2904641366 abstract "DNA variation cannot explain all trait variance in chronic pain conditions. Altered epigenetic marks as a result of gene-environment interactions are additional factors to be considered in pain genetics. Environmental factors such as stress, nerve and tissue injuries, and disease conditions impact on the vulnerability of individuals to develop chronic pain, likely via DNA sequence-independent mechanisms. Pursuing epigenetic alterations in chronic pain will help to find potential biomarkers and direct specific treatment strategies. This chapter will overview evidence mainly from clinical studies on altered genetic marks, including DNA methylation, histone code, and noncoding RNAs, in a variety of chronic pain conditions. We will also discuss the outcome of human studies on a number of painful disease conditions where pain was not directly assessed, since the epigenetic marks identified in those studies may help further epigenetic studies in the context of chronic pain." @default.
- W2904641366 created "2018-12-22" @default.
- W2904641366 creator A5016029838 @default.
- W2904641366 creator A5067073623 @default.
- W2904641366 date "2019-01-01" @default.
- W2904641366 modified "2023-09-23" @default.
- W2904641366 title "An Overview of Epigenetic Correlates of Human Chronic Pain Conditions" @default.
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