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• W2905343313 abstract "Asthma has long been considered to be driven by allergen-specific T helper 2 (Th2) responses. Although Th2 cytokines produced by Th2 cells play a central role in the induction and regulation of airway inflammation, clinical and basic studies have begun to shed light on the role of many types of cells. It is now well recognized that asthma is a multicellular disease, involving abnormal responses of many different cell types in the lung, including airway structural cells and innate and adaptive immune cells. A subpopulation of CD4+ T cells, such as Th1, Th17, and regulatory T cells (Tregs), has also been implicated in the regulation of airway inflammation. Airway epithelial cells and dendritic cells play an important role in promoting innate and adaptive immune responses in asthma. Group 2 innate lymphoid cells (ILC2s) stimulated by epithelial cell-derived cytokines, IL-25, IL-33, and thymic stromal lymphopoietin (TSLP), produce Th2 cytokines and have been reported to be involved in the induction of airway inflammation. Complementary experimental approaches including cultured cells, animal models, and human clinical studies have provided many insights into diverse cellular mechanisms in this complex disease." @default.
• W2905343313 created "2018-12-22" @default.
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• W2905343313 date "2018-12-19" @default.
• W2905343313 modified "2023-09-24" @default.
• W2905343313 title "Cellular Mechanisms of Allergic Airway Inflammation" @default.
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• W2905343313 doi "https://doi.org/10.1007/978-981-13-2790-2_3" @default.
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