Matches in SemOpenAlex for { <https://semopenalex.org/work/W2906245669> ?p ?o ?g. }
- W2906245669 endingPage "7" @default.
- W2906245669 startingPage "7" @default.
- W2906245669 abstract "Autophagy, an intracellular degradation mechanism, has many immunological functions and is a constitutive process necessary for maintaining cellular homeostasis and organ structure. One of the functions of autophagy is to control the innate immune response. Many studies conducted in recent years have revealed the contribution of autophagy to the innate immune response, and relationships between this process and various diseases have been reported. Inflammatory bowel disease is an intractable disorder with unknown etiology; however, immunological abnormalities in the intestines are known to be involved in the pathology of inflammatory bowel disease, as is dysfunction of autophagy. In Crohn’s disease, many associations with autophagy-related genes, such as ATG16L1, IRGM, NOD2, and others, have been reported. Abnormalities in the ATG16L1 gene, in particular, have been reported to cause autophagic dysfunction, resulting in enhanced production of inflammatory cytokines by macrophages as well as abnormal function of Paneth cells, which are important in intestinal innate immunity. In this review, we provide an overview of the autophagy mechanism in innate immune cells in inflammatory bowel disease." @default.
- W2906245669 created "2019-01-01" @default.
- W2906245669 creator A5000086080 @default.
- W2906245669 creator A5002391399 @default.
- W2906245669 creator A5023577820 @default.
- W2906245669 creator A5024419028 @default.
- W2906245669 creator A5032866222 @default.
- W2906245669 date "2018-12-22" @default.
- W2906245669 modified "2023-10-01" @default.
- W2906245669 title "Impact of Autophagy of Innate Immune Cells on Inflammatory Bowel Disease" @default.
- W2906245669 cites W1014257459 @default.
- W2906245669 cites W1488686583 @default.
- W2906245669 cites W1523693806 @default.
- W2906245669 cites W1529674879 @default.
- W2906245669 cites W1561041814 @default.
- W2906245669 cites W1574582813 @default.
- W2906245669 cites W1588546052 @default.
- W2906245669 cites W1642457845 @default.
- W2906245669 cites W1650516212 @default.
- W2906245669 cites W1670573097 @default.
- W2906245669 cites W1803970038 @default.
- W2906245669 cites W1827069369 @default.
- W2906245669 cites W1894136033 @default.
- W2906245669 cites W1925042390 @default.
- W2906245669 cites W1926674024 @default.
- W2906245669 cites W1934000495 @default.
- W2906245669 cites W1942767863 @default.
- W2906245669 cites W1951818427 @default.
- W2906245669 cites W1966342688 @default.
- W2906245669 cites W1971046821 @default.
- W2906245669 cites W1976056371 @default.
- W2906245669 cites W1979318608 @default.
- W2906245669 cites W1983251816 @default.
- W2906245669 cites W1985538633 @default.
- W2906245669 cites W1987493079 @default.
- W2906245669 cites W1994823455 @default.
- W2906245669 cites W1996068251 @default.
- W2906245669 cites W1996762292 @default.
- W2906245669 cites W2001045944 @default.
- W2906245669 cites W2003800301 @default.
- W2906245669 cites W2004111402 @default.
- W2906245669 cites W2004608617 @default.
- W2906245669 cites W2009523990 @default.
- W2906245669 cites W2014913358 @default.
- W2906245669 cites W2021178651 @default.
- W2906245669 cites W2024211292 @default.
- W2906245669 cites W2025511820 @default.
- W2906245669 cites W2026563152 @default.
- W2906245669 cites W2028536701 @default.
- W2906245669 cites W2032227357 @default.
- W2906245669 cites W2036109191 @default.
- W2906245669 cites W2042260827 @default.
- W2906245669 cites W2042767581 @default.
- W2906245669 cites W2043724674 @default.
- W2906245669 cites W2043775908 @default.
- W2906245669 cites W2044676100 @default.
- W2906245669 cites W2045434069 @default.
- W2906245669 cites W2047208232 @default.
- W2906245669 cites W2047341425 @default.
- W2906245669 cites W2047923753 @default.
- W2906245669 cites W2048030072 @default.
- W2906245669 cites W2050449334 @default.
- W2906245669 cites W2051173337 @default.
- W2906245669 cites W2051303794 @default.
- W2906245669 cites W2064741210 @default.
- W2906245669 cites W2065031087 @default.
- W2906245669 cites W2065864183 @default.
- W2906245669 cites W2071025253 @default.
- W2906245669 cites W2074784443 @default.
- W2906245669 cites W2075708010 @default.
- W2906245669 cites W2076062022 @default.
- W2906245669 cites W2077417036 @default.
- W2906245669 cites W2078763748 @default.
- W2906245669 cites W2080875750 @default.
- W2906245669 cites W2080901528 @default.
- W2906245669 cites W2084708009 @default.
- W2906245669 cites W2084802884 @default.
- W2906245669 cites W2085828546 @default.
- W2906245669 cites W2093356677 @default.
- W2906245669 cites W2095347614 @default.
- W2906245669 cites W2096290681 @default.
- W2906245669 cites W2096853390 @default.
- W2906245669 cites W2100067355 @default.
- W2906245669 cites W2102424853 @default.
- W2906245669 cites W2105409495 @default.
- W2906245669 cites W2106348311 @default.
- W2906245669 cites W2106855445 @default.
- W2906245669 cites W2107480391 @default.
- W2906245669 cites W2109070574 @default.
- W2906245669 cites W2109491705 @default.
- W2906245669 cites W2118300504 @default.
- W2906245669 cites W2120511085 @default.
- W2906245669 cites W2123123035 @default.
- W2906245669 cites W2127920553 @default.
- W2906245669 cites W2128724486 @default.
- W2906245669 cites W2132763961 @default.
- W2906245669 cites W2138694950 @default.
- W2906245669 cites W2140267261 @default.