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• W2906799341 abstract "Mitochondria can no longer be considered as static structures whose sole function is the unobtrusive manufacture of ATP. It is now clear that they also represent a storeroom for a number of potentially lethal proteins that are unleashed during programmed cell death and that they are significant participants in the detailed intracellular organization of cellular [Ca 2+ ] c signaling. While the expression in the mitochondrial membrane of Ca 2+ transporting mechanisms was established years ago, the physiological significance of these pathways has only recently become apparent. There is now no question that mitochondria will take up and accumulate Ca 2+ in all cells studied during the routine events of cellular [Ca 2+ ] c signaling, and that the pathway influences both mitochondrial function itself and the spatiotemporal and quantitative characteristics of the cellular [Ca 2+ ] c signal. As general issues relating to mitochondrial Ca 2+ handling have recently been widely reviewed this chapter proposes to highlight some of the more controversial and novel developments in the field over recent years and some of the mechanistic, quantitative, and comparative questions that remain." @default.
• W2906799341 created "2019-01-11" @default.
• W2906799341 creator A5018410798 @default.
• W2906799341 date "2003-01-01" @default.
• W2906799341 modified "2023-09-23" @default.
• W2906799341 title "Mitochondria and Calcium Signaling, Point and Counterpoint" @default.
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• W2906799341 doi "https://doi.org/10.1016/b978-012124546-7/50496-4" @default.
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