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- W2907813553 abstract "The target of this animal study was to clarify the influence of Copper oxychloride (COC) (at concentrations of 50, 100 and 200 mg/kg b.wt.) administration for ninety days on the brain tissues to evaluate its possible neurotoxicity.Thirty male albino rats were divided up into control and four experimental groups. Group-II (rats were fed corn oil daily through oral gavage) and Group-III-A, Group-III-B, Group-III-C (rats were fed orally with COC in a dosage of 50 mg/kg, 100 mg/kg and 200 mg/kg b.wt., respectively, daily for ninety days. Various biochemical analyses and histopathological assessment of rat forebrain were investigated.the brains of the treated rats at the three chosen doses of COC recorded a significant (p≤0.05) elevation of lipid peroxidation. The measured brain lactate dehydrogenase (LDH) revealed non significant (p ≥ 0.05) differences among the studied groups. Besides, there was a significant (p < 0.05) decrease in the brain manganese concentration (Mn) of COC treated rats. In addition, there were significant (p< 0.05) increase in zinc (Zn) brain concentration and non significant change in copper (Cu) brain concentration among groups. The brain, cerebrum showed marked histopathological damage than cerebellum. The cerebral cortex of COC treated animals exhibited severe degenerative changes.The present results concluded that consumption of food contaminated even with modest amount of COC can enter the brain barrier resulted in neurotoxicity in the brain of albino rats." @default.
- W2907813553 created "2019-01-11" @default.
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- W2907813553 date "2019-03-01" @default.
- W2907813553 modified "2023-10-18" @default.
- W2907813553 title "The neurotoxic impact of subchronic exposure of male rats to copper oxychloride" @default.
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- W2907813553 doi "https://doi.org/10.1016/j.jtemb.2018.12.015" @default.
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