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- W2909260600 abstract "This chapter presents the three different strategies for achieving cardioprotection and saving the patients from deleterious ischemic damages. The first strategy is to increase ischemic tolerance before the onset of myocardial ischemia. This strategy is effective for patients with coronary artery disease or coronary risk factors. Preventing rupture of the atheroma is another paradigm used to attenuate the incidence of acute coronary syndrome. HMG-CoAreductase inhibitors, such as pravastatin may attenuate plaque rupture and may decrease the incidence of cardiac death in patients with coronary artery disease or in high-risk patients. Growth factors can induce collateral circulation to prevent or attenuate the ischemic damages. Finding the trigger mechanisms of the infarct size-limiting effect of ischemic preconditioning is important in inducing ischemic tolerance in advance. Another method used to increase ischemic tolerance is to induce cardioprotection prior to the ischemia of acute myocardial infarction, and one potential way is to induce in advance the infarct size-limiting effect of ischemic preconditioning in patients with coronary artery disease. The second strategy is to attenuate ischemia and reperfusion injury when the irreversible process of myocardial cellular injury occurs. Pharmacological interventions, such as adenosine or nitric oxide, may contribute to attenuate the ischemic damages. The third strategy is to treat ischemic chronic heart failure that is caused after acute myocardial infarction. Gene therapy or the development of artificial hearts may provide a potential treatment in chronic failing hearts." @default.
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- W2909260600 date "2001-01-01" @default.
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- W2909260600 title "Cellular Mechanisms of Cardioprotection" @default.
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- W2909260600 doi "https://doi.org/10.1016/b978-012656975-9/50051-1" @default.
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