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- W2910439306 abstract "Streptococcus gallolyticus is an opportunistic pathogen responsible for septicemia and endocarditis in the elderly and is also strongly associated with colorectal cancer. S. gallolyticus can form biofilms, express specific pili to colonize the host tissues, and produce a specific bacteriocin allowing killing of commensal bacteria in the murine colon. Nevertheless, how the expression of these colonization factors is regulated remains largely unknown. Here, we show that c-di-AMP plays pleiotropic roles in S. gallolyticus , controlling the tolerance to osmotic stress, cell size, biofilm formation on abiotic surfaces, adherence and cell aggregation on human intestinal cells, expression of Pil3 pilus, and production of bacteriocin. This study indicates that c-di-AMP may constitute a key regulatory molecule for S. gallolyticus host colonization and pathogenesis." @default.
- W2910439306 created "2019-01-25" @default.
- W2910439306 creator A5004988031 @default.
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- W2910439306 creator A5031476041 @default.
- W2910439306 creator A5078350659 @default.
- W2910439306 date "2019-03-15" @default.
- W2910439306 modified "2023-09-25" @default.
- W2910439306 title "Increased Intracellular Cyclic di-AMP Levels Sensitize Streptococcus gallolyticus subsp. gallolyticus to Osmotic Stress and Reduce Biofilm Formation and Adherence on Intestinal Cells" @default.
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- W2910439306 doi "https://doi.org/10.1128/jb.00597-18" @default.
- W2910439306 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6398277" @default.
- W2910439306 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/30617242" @default.
- W2910439306 hasPublicationYear "2019" @default.
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