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- W2912245621 endingPage "14472" @default.
- W2912245621 startingPage "14460" @default.
- W2912245621 abstract "Phosphoinositide 3-kinase (PI3K), a crucial signaling molecule, is regulated by various upstream regulators. Traditionally, receptor tyrosine kinases and G protein-coupled receptor are regarded as its principle upstream regulators; however, recent reports have indicated that spleen tyrosine kinase, β-arrestin2, Janus kinase, and RAS can also perform this role. Dysregulation of PI3K is common in the progression of various diseases, including, but not limited to, tumors, Alzheimer's disease, Parkinson's disease, rheumatoid arthritis, and acute myelogenous leukemia. The aim of this review is to provide a perspective on PI3K-related diseases examining both the classical and nonclassical upstream regulators of PI3K in detail." @default.
- W2912245621 created "2019-02-21" @default.
- W2912245621 creator A5003799076 @default.
- W2912245621 creator A5013419645 @default.
- W2912245621 creator A5017440852 @default.
- W2912245621 creator A5068990370 @default.
- W2912245621 date "2019-02-02" @default.
- W2912245621 modified "2023-10-12" @default.
- W2912245621 title "Upstream regulators of phosphoinositide 3‐kinase and their role in diseases" @default.
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