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- W2912382068 abstract "Post-transplant lymphoproliferative disorders (PTLDs) cover a broad spectrum of lymphoproliferative lesions arising after solid organ or allogeneic hematopoietic stem cell transplantation. The composition and function of the tumor microenvironment (TME), consisting of all non-malignant constituents of a tumor, is greatly impacted in PTLD through a complex interplay between 4 factors: 1) the graft organ causes immune stimulation through chronic antigen presentation; 2) the therapy to prevent organ rejection interferes with the immune system; 3) the oncogenic Epstein-Barr virus (EBV), present in 80% of PTLDs, has a causative role in the oncogenic transformation of lymphocytes and influences immune responses; 4) interaction with the donor-derived immune cells accompanying the graft. These factors make PTLDs an interesting model to look at cancer-microenvironment interactions and current findings can be of interest for other malignancies including solid tumors. Here we will review the current knowledge of the TME composition in PTLD with a focus on the different factors involved in PTLD development." @default.
- W2912382068 created "2019-02-21" @default.
- W2912382068 creator A5009283491 @default.
- W2912382068 creator A5086292280 @default.
- W2912382068 date "2019-01-24" @default.
- W2912382068 modified "2023-10-12" @default.
- W2912382068 title "The Tumor Microenvironment in Post-Transplant Lymphoproliferative Disorders" @default.
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