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• W2912485812 abstract "Several studies have proposed cerebral malaria (CM) as a CD4+ and CD8+ T lymphocyte-mediated disease. However, there are no data regarding the recruitment and/or persistence of these cells in the CNS following the phase of infection resolution. Glutamate-mediate excitotoxicity has also been implicated in CM. Blockade of glutamate NMDA receptors by its noncompetitive antagonist MK801 modulates cytokine and neurotrophic factors expression preventing cognitive and depressive-like behavior in experimental CM. Herein, we aim to investigate the role of T lymphocytes in later outcomes in CM, and whether the protective role of MK801 is associated with T lymphocytes response." @default.
• W2912485812 created "2019-02-21" @default.
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• W2912485812 date "2019-05-01" @default.
• W2912485812 modified "2023-09-27" @default.
• W2912485812 title "T-lymphocytes response persists following Plasmodium berghei strain Anka infection resolution and may contribute to later experimental cerebral malaria outcomes" @default.
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• W2912485812 doi "https://doi.org/10.1016/j.jneuroim.2019.02.002" @default.
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