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- W291256670 abstract "The α2-adrenoceptor agonists clonidine (0.03 μM) and St 363 (10 μM) inhibited electrically-stimulated noradrenaline release from mouse isolated atria incubated in [3H]-noradrenaline, and the α2-adrenoceptor antagonist idazoxan (0.1 μM) enhanced noradrenaline release. None of the effects of these drugs were altered by pretreatment of the mice with pertussis toxin (1.5 μg/mouse). In slices of rat kidney cortex, clonidine (0.1 μM) and the α1-adrenoceptor agonist methoxamine (10 μM) inhibited electrically-stimulated noradrenaline release, and idazoxan (0.1 μM) enhanced noradrenaline release. Pertussis toxin treatment (25 μg/kg) abolished the inhibitory effect of methoxamine, but did not alter the effects of clonidine or idazoxan. These results suggest that inhibitory prejunctional α1-adrenoceptors function through a pertussis toxin-sensitive G-protein, whereas prejunctional α2-adrenoceptors do not." @default.
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- W291256670 date "1991-01-01" @default.
- W291256670 modified "2023-10-16" @default.
- W291256670 title "G-proteins and Prejunctional α-adrenoceptors" @default.
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- W291256670 doi "https://doi.org/10.1016/b978-0-08-041165-1.50092-2" @default.
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