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- W2912571569 abstract "Despite years of significant effort, it remains unclear how injured cardiomyocytes disrupt gene synthesis in adipocytes. We recently demonstrate cardiac ischemia/reperfusion (I/R) injury markedly releases exosomes. In our current discovery-based study, we determined the specific effect of cardiac-derived exosomes upon gene expression. By investigating genes regulating various cellular processes (including inflammation, lipid biosynthesis, lipolysis, glycometabolism, β oxidation, adipokine, apoptosis and ER stress), we demonstrate that cardiac-derived exosomes upregulated 1 cytokine and 5 ER stress genes (CHOP, Gadd34), and decreased the expression of 2 ER stress genes (EDEM1, DERL1) in adipose tissue in vivo. To further investigate the dysregulation of ER stress, ERp44 and DsBA-L1 was measured. Cardiac I/R-derived exosomes suppressed expression of both ERp44 and DsBA-L1 in vitro and in vivo, supporting the role of exosome-induced ER stress in target cells. In 3T3L1 cells subjected to cardiac I/R-derived e..." @default.
- W2912571569 created "2019-02-21" @default.
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- W2912571569 date "2017-11-14" @default.
- W2912571569 modified "2023-10-07" @default.
- W2912571569 title "Abstract 16449: Exosome-Derived miRNAs Mediate Cardiomyocyte-Adipocyte Communication and Suppress High Molecule Adiponectin Production by Regulating ER Function After Myocardial Ischemia/Reperfusion" @default.
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