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- W2912662942 abstract "Androgen deprivation therapy for prostate cancer (PCa) benefits patients with early disease, but becomes ineffective as PCa progresses to a castration-resistant state (CRPC). Initially CRPC remains dependent on androgen receptor (AR) signaling, often through increased expression of full-length AR (ARfl) or expression of dominantly active splice variants such as ARv7. We show in ARv7-dependent CRPC models that ARv7 binds together with ARfl to repress transcription of a set of growth-suppressive genes. Expression of the ARv7-repressed targets and ARv7 protein expression are negatively correlated and predicts for outcome in PCa patients. Our results provide insights into the role of ARv7 in CRPC and define a set of potential biomarkers for tumors dependent on ARv7." @default.
- W2912662942 created "2019-02-21" @default.
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- W2912662942 date "2019-03-01" @default.
- W2912662942 modified "2023-10-16" @default.
- W2912662942 title "ARv7 Represses Tumor-Suppressor Genes in Castration-Resistant Prostate Cancer" @default.
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