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- W2912776042 abstract "Transcranial alternating current stimulation (tACS) modulates neural oscillatiory activity and excitability in the primary motor cortex (M1). Previous studies have found that these effects can last for more than an hour and an involvement of N-methyl-D-aspartate receptor (NMDAR) mediated synaptic plasticity has been suggested. However, the cortical mechanisms underlying tACS after-effects are not fully understood. In the present study 20 Hz beta tACS was applied to M1 after participants received either an NMDAR antagonist (dextromethorphan) or a placebo and the effects on cortical beta oscillations and excitability were explored. In separate sessions the effects of the pharmacological intervention were investigated on a 4-minute resting state electroencephalogram (EEG) and on M1 excitability measured by single and paired pulse transcranial magnetic stimulation (TMS). Results showed that after placebo medication, beta tACS induced an increase in cortical excitability and beta oscillations for at least 60 minutes However, when dextromethorphan was administered, these effects were completely abolished. No effects of tACS on paired pulse TMS measures were found. These results provide causal evidence for tACS-induced NMDAR-mediated plasticity in the motor cortex, which advances our understanding of tACS-mediated influences on human motor cortex physiology." @default.
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- W2912776042 date "2019-03-01" @default.
- W2912776042 modified "2023-09-25" @default.
- W2912776042 title "Motor cortex plasticity induced by beta transcranial alternating current stimulation" @default.
- W2912776042 doi "https://doi.org/10.1016/j.brs.2018.12.448" @default.
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