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- W2912851989 abstract "Introduction: Emerging evidence implicates crosstalks between endoplasmic reticulum (ER) stress and oxidative stress in regulating pathogenesis of heart disease. At one arm, elevated ROS is reported to mediate reversible oxidation of the ER stress subproteome, altering their regulatory impact in cardiac remodeling; in conjunction, a cluster of ER stress proteins is found responsible to maintain homeostasis of the redox subproteome. However, three critical questions remain: (i) which of the ER stress proteins are targets of oxidative stress modification, (ii) what are the sites of their modification, and (iii) how is this process dynamically regulated. Methods: We have taken a targeted quantitative proteomics approach incorporating biotin-switch and isobaric labeling methods to determine proteome properties (expression, turnover, and PTM) of ER stress proteins in sham or ISO-challenged C57BL6/J mouse heart at 6 time points: Day 1, 3, 5, 7, 10, and 14. In parallel, functional evaluations (e.g., HW/BW) were ..." @default.
- W2912851989 created "2019-02-21" @default.
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- W2912851989 date "2017-11-14" @default.
- W2912851989 modified "2023-09-27" @default.
- W2912851989 title "Abstract 16977: Characterization of Oxidative Stress Regulation of Endoplasmic Reticulum Stress Pathway in Cardiac Remodeling" @default.
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