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- W2912934029 endingPage "231" @default.
- W2912934029 startingPage "231" @default.
- W2912934029 abstract "Many studies have shown that low flux nitric oxide (NO) produced by inducible NO synthase (iNOS/NOS2) in various tumors, including glioblastomas, can promote angiogenesis, cell proliferation, and migration/invasion. Minimally invasive, site-specific photodynamic therapy (PDT) is a highly promising anti-glioblastoma modality. Recent research in the authors’ laboratory has revealed that iNOS-derived NO in glioblastoma cells elicits resistance to 5-aminolevulinic acid (ALA)-based PDT, and moreover endows PDT-surviving cells with greater proliferation and migration/invasion aggressiveness. In this contribution, we discuss iNOS/NO antagonism to glioblastoma PDT and how this can be overcome by judicious use of pharmacologic inhibitors of iNOS activity or transcription." @default.
- W2912934029 created "2019-02-21" @default.
- W2912934029 creator A5045290396 @default.
- W2912934029 creator A5065281865 @default.
- W2912934029 date "2019-02-15" @default.
- W2912934029 modified "2023-10-16" @default.
- W2912934029 title "Nitric Oxide Antagonism to Anti-Glioblastoma Photodynamic Therapy: Mitigation by Inhibitors of Nitric Oxide Generation" @default.
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- W2912934029 doi "https://doi.org/10.3390/cancers11020231" @default.
- W2912934029 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6406633" @default.
- W2912934029 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/30781428" @default.
- W2912934029 hasPublicationYear "2019" @default.
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