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- W2912995162 endingPage "119" @default.
- W2912995162 startingPage "87" @default.
- W2912995162 abstract "This chapter presents a summary of normal anatomy of the liver, frequent anomalies, embryology, physiologic correlates, and anatomic abnormalities found in cirrhosis. The branching pattern of veins defines hepatic segments. Arterial injury or ligation may lead to ischemic strictures of the ducts. The liver receives most of the splanchnic blood flow. After severe obstruction at any level of the hepatic vasculature, collateral channels become a source of bleeding and contribute to hepatic encephalopathy. The microvasculature defines the parenchymal subunits called acini or lobules. Hepatocytes are exposed to gradients of nutrients and waste products leading to zonal variation in susceptibility to ischemia and drug toxicity. Sinusoidal endothelial cells are fenestrated to facilitate rapid exchange between plasma and hepatocytes. Hepatic lymph accumulates as ascites in response to increased sinusoidal pressure. Cirrhosis occurs when obliteration of small portal and hepatic veins causes local parenchymal extinction lesions that collapse to form septa." @default.
- W2912995162 created "2019-02-21" @default.
- W2912995162 creator A5002443282 @default.
- W2912995162 date "2011-10-31" @default.
- W2912995162 modified "2023-09-27" @default.
- W2912995162 title "Physioanatomic Considerations" @default.
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