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• W2913426274 abstract "Significance Effector Treg cells comprise the subset of the Treg cell population that exhibits enhanced regulatory function. Whereas the induction and maintenance of this subset are known to depend on TCR signaling, the underlying molecular mechanisms downstream of such signaling and the contributions of individual TCR-dependent genes to effector Treg cell generation are still poorly understood. In the studies described here differentiated Treg cells in which E-protein (E2A/HEB) expression has been deleted were utilized to demonstrate that E proteins are transcriptional suppressors of a large number of genes associated with effector Treg cell differentiation, localization, function, and proliferation. Thus, this finding indicates that continuous TCR signals modulating E-protein activity is a major mechanism underlying Treg cell acquisition of effector functions." @default.
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• W2913426274 date "2019-02-15" @default.
• W2913426274 modified "2023-10-16" @default.
• W2913426274 title "E-protein regulatory network links TCR signaling to effector Treg cell differentiation" @default.
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• W2913426274 doi "https://doi.org/10.1073/pnas.1800494116" @default.
• W2913426274 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6410771" @default.
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