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- W2918126986 abstract "Background Our previous work has demonstrated that treatment with isoflurane has a preconditioning-like inhibitory effect on superoxide production (SOP) by polymorphonuclear neutrophils. The current objectives were to determine persistency of this effect and to clarify where in the signalling pathway this inhibition of SOP occurred. The latter was accomplished using two receptor-dependent neutrophil agonists, platelet activating factor (PAF) and formyl-methionyl-leucyl-phenylalanine (fMLP), and two receptor-independent neutrophil stimuli, the protein-kinase C stimulator, phorbol myristate acetate (PMA), and the calcium ionophore, A23187. Methods Arterial blood samples were obtained from eight dogs under baseline condition (conscious state), during isoflurane (1 MAC) administration, and 24 and 48 hr post-isoflurane (also in conscious state). Neutrophils were isolated and stimulated with 1 lM concentrations of PAF, fMLP, PMA, and A23187. SOP was measured spectrophotometrically." @default.
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- W2918126986 date "2010-01-01" @default.
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- W2918126986 title "Persistency and pathway of isoflurane-induced inhibition of superoxide production by neutrophils Persistance et voie de l'inhibition de la production de superoxyde par les neutrophiles induite par isoflurane" @default.
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