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• W2919975746 startingPage "1671" @default.
• W2919975746 abstract "Abstract Astrocytes generate robust intracellular Ca 2+ concentration changes (Ca 2+ signals), which are assumed to regulate astrocytic functions that play crucial roles in the regulation of brain functions. One frequently used strategy for exploring the role of astrocytic Ca 2+ signalling is the use of mice deficient in the type 2 inositol 1,4,5‐trisphosphate receptor (IP 3 R2). These IP 3 R2‐knockout (KO) mice are reportedly devoid of Ca 2+ mobilization from the endoplasmic reticulum (ER) in astrocytes. However, they have shown no functional deficits in several studies, causing a heated debate as to the functional relevance of ER‐mediated Ca 2+ signalling in astrocytes. Recently, the assumption that Ca 2+ mobilization from the ER is absent in IP 3 R2‐KO astrocytes has been re‐evaluated using intraorganellar Ca 2+ imaging techniques. The new results indicated that IP 3 R2‐independent Ca 2+ release may generate Ca 2+ nanodomains around the ER, which may help explain the absence of functional deficits in IP 3 R2‐KO mice. image" @default.
• W2919975746 created "2019-03-11" @default.
• W2919975746 creator A5009491639 @default.
• W2919975746 creator A5042349107 @default.
• W2919975746 date "2019-03-18" @default.
• W2919975746 modified "2023-10-15" @default.
• W2919975746 title "Visualization of astrocytic intracellular Ca ²⁺ mobilization" @default.
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