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- W2922081722 abstract "Clioquinol, one of the first mass-produced drugs, was considered safe and efficacious for many years. It was used as an antifungal and an antiprotozoal drug until it was linked to an outbreak of subacute myelo-optic neuropathy (SMON), a debilitating disease almost exclusively confined to Japan. Today, new information regarding clioquinol targets and its mechanism of action, as well as genetic variation (SNPs) in efflux transporters in the Japanese population, provide a unique interpretation of the existing phenomena. Further understanding of clioquinol's role in the inhibition of cAMP efflux and promoting apoptosis might offer promise for the treatment of cancer and/or neurodegenerative diseases. Here, we highlight recent developments in the field and discuss possible connections, hypotheses and perspectives in clioquinol-related research." @default.
- W2922081722 created "2019-03-22" @default.
- W2922081722 creator A5028187863 @default.
- W2922081722 creator A5071867144 @default.
- W2922081722 creator A5079040495 @default.
- W2922081722 date "2019-07-01" @default.
- W2922081722 modified "2023-09-23" @default.
- W2922081722 title "Clioquinol: To harm or heal" @default.
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- W2922081722 doi "https://doi.org/10.1016/j.pharmthera.2019.03.009" @default.
- W2922081722 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6571072" @default.
- W2922081722 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/30898518" @default.
- W2922081722 hasPublicationYear "2019" @default.
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