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- W2922308488 abstract "L’existence de protocoles très divers d’immunothérapie spécifique (IT), par voies sous-cutanée, sublinguale, orale, intra-lymphatique ou épicutanée, reflète l’absence de modèle simple expliquant l’action de ces thérapeutiques. La somme des travaux antérieurs permet cependant d’identifier les éléments cellulaires clés de l’IT, que sont les cellules dendritiques tolérogènes, les lymphocytes T régulateurs Foxp3+ et/ou producteurs de cytokines immunosuppressives (IL-10, TGFβ), et les lymphocytes B régulateurs, producteurs d’IL-10 et susceptibles de se différencier en plasmocytes à IgG4. Ces éléments régulateurs doivent agir sur les cellules amplificatrices (ILC2) et effectrices (T effecteurs, mastocytes, basophiles) des réponses Th2 afin de réaliser le « défi immunologique » que représente la suppression ou la déviation de ces réponses.The existence of diverse protocols of specific immunotherapy (IT), namely SCIT, SLIT, OIT, intra-lymphatic and EPIT, reflects the absence of a simple model explaining the mode of action of these therapies. Numerous previous studies, however, makes it possible to identify the key cellular elements of IT, which are: tolerogenic dendritic cells, Foxp3 + regulatory T lymphocytes producing immunosuppressive cytokines (IL-10, TGFβ), and IL-10+ regulatory B cells, capable to differentiate into IgG4 plasma cells. These regulatory elements must act on the amplifying (ILC2) and effector (effector T lymphocytes, mast cells, basophils) actors of the Th2 responses, in order to induce the immunological challenge represented by the suppression or deviation of these responses." @default.
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- W2922308488 date "2019-04-01" @default.
- W2922308488 modified "2023-10-17" @default.
- W2922308488 title "Œsophagite à éosinophiles : le point sur les consensus de prise en charge" @default.
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- W2922308488 doi "https://doi.org/10.1016/j.reval.2019.02.216" @default.
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