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• W2930194799 abstract "Prenatal nicotine exposure (PNE) is closely related to depression in offspring. However, the underlying mechanism is still unclear. We hypothesized that neurosteroid in the hippocampus may mediate PNE‑induced depression‑like behaviors. Nicotine was subcutaneously administered (1.0 mg/kg) to pregnant rats twice daily from gestational day (GD) 9 to 20. In adolescent offspring, PNE significantly increased immobility time and decreased the sucrose preference in female rats. The numbers of hippocampal neurons declined in the CA3 and DG regions. Steroidogenic acute regulatory protein (StAR) expression was suppressed in female rats. In fetal offspring, the neuronal numbers of CA3 regions in PNE female fetal hippocampal were significantly decreased, accompanied by the enhanced content of corticosterone and StAR expression. These data indicated that PNE induced depression‑like behavior in adolescent female rats via the regulation of neurosteroid levels in the hippocampus." @default.
• W2930194799 created "2019-04-11" @default.
• W2930194799 creator A5011814799 @default.
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• W2930194799 date "2019-03-29" @default.
• W2930194799 modified "2023-10-09" @default.
• W2930194799 title "Prenatal nicotine exposure induces depression‑like behavior in adolescent female rats via modulating neurosteroid in the hippocampus" @default.
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• W2930194799 doi "https://doi.org/10.3892/mmr.2019.10105" @default.
• W2930194799 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6471439" @default.
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