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- W2934655650 abstract "To sustain life in environments that are fraught with risks of life-threatening injury, organisms have developed innate protective strategies such that the response to wounds is rapid and localized, with the simultaneous recruitment of molecular, biochemical and cellular pathways that limit bleeding and eliminate pathogens and damaged host cells, while promoting effective healing. These pathways are tightly regulated, as their over- or under-activation may lead to inadequate healing, disease, and/or demise of the host. Recent advances in our understanding of coagulation and complement, a key component of innate immunity, have revealed critical molecular links that regulate the delicate balance of both systems, thereby providing the necessary elements to achieve homeostasis. Polyphosphate is one such factor that intriguingly, plays a key role by promoting coagulation, while dampening complement activation, a discovery that is uncovering novel insights into mechanisms underlying disease and new therapeutic strategies." @default.
- W2934655650 created "2019-04-11" @default.
- W2934655650 creator A5068650400 @default.
- W2934655650 date "2019-04-04" @default.
- W2934655650 modified "2023-09-26" @default.
- W2934655650 title "Polyphosphates and Complement Activation" @default.
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- W2934655650 doi "https://doi.org/10.3389/fmed.2019.00067" @default.
- W2934655650 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6458250" @default.
- W2934655650 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/31019911" @default.
- W2934655650 hasPublicationYear "2019" @default.
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