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• W2936233025 abstract "Acne vulgaris is a common inflammatory skin disorder which is recognizable by dermatological lesions and scars. In addition to some pathogenetic factors such as hyperkeratinization, upregulated sebum secretion, and immunoinflammatory reactions, recent studies have also connected stress to the pathogenesis of acne vulgaris. In this article, we will briefly review clinical studies that interrogated alterations in stress biomarkers by a systematic search conducted in PubMed, Web of Science, and Scopus using acne, oxidative stress, and oxygen keywords. Overall, studies have shown that biomarkers (e.g. lipid peroxidation final products) are higher in acne vulgaris lesions. A significant positive correlation has also been noted between acne severity and biomarkers. In contrast, diminished levels of antioxidant enzymes (e.g. superoxide dismutase and catalase) have been observed in acne. We propose four probable mechanisms for the role of reactive oxygen species (ROS) in acne pathogenesis. We believe that ROS can contribute significantly to the acne vulgaris pathobiology via toll-like receptor (TLR), peroxisome proliferator-activated receptor (PPAR), mTOR pathway, and innate immune system, resulting in inflammation by alterations in the generation of several proinflammatory cytokines including IL-1, IL-8, and TNF-α." @default.
• W2936233025 created "2019-04-25" @default.
• W2936233025 creator A5023956394 @default.
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• W2936233025 date "2019-04-10" @default.
• W2936233025 modified "2023-10-03" @default.
• W2936233025 title "Evidence for the Important Role of Oxidative Stress in the Pathogenesis of Acne." @default.
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• W2936233025 doi "https://doi.org/10.31661/gmj.v0i0.1291" @default.
• W2936233025 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/8344136" @default.
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• W2936233025 hasPublicationYear "2019" @default.
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