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• W2941393885 abstract "Epstein-Barr virus (EBV) immortalization of resting B lymphocytes (RBLs) is a useful model system to study EBV oncogenesis. By incorporating transcriptome sequencing (RNA-seq), chromatin immune precipitation followed by deep sequencing (ChIP-seq), chromatin interaction analysis followed by paired-end tag sequencing (ChIA-PET), and genome-wide clustered regularly interspaced short palindromic repeat (CRISPR) screen, we identified key pathways that EBV usurps to enable B cell growth and transformation. Multiple layers of regulation could be achieved by cooperations between multiple EBV transcription factors binding to the same enhancers. EBV manipulated the expression of most cell genes essential for lymphoblastoid cell line (LCL) growth and survival. In addition to proteins, long noncoding RNAs (lncRNAs) regulated by EBV also contributed to LCL growth and survival. The data presented in this paper not only allowed us to further define the molecular pathogenesis of EBV but also serve as a useful resource to the EBV research community." @default.
• W2941393885 created "2019-05-03" @default.
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• W2941393885 date "2019-07-01" @default.
• W2941393885 modified "2023-10-16" @default.
• W2941393885 title "RNA Sequencing Analyses of Gene Expression during Epstein-Barr Virus Infection of Primary B Lymphocytes" @default.
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• W2941393885 doi "https://doi.org/10.1128/jvi.00226-19" @default.
• W2941393885 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6580941" @default.
• W2941393885 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/31019051" @default.
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