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- W2942172892 abstract "Ascending thoracic aortic aneurysm (TAA) develops in about 35% of subjects with bicuspid aortic valve (BAV). It is a congenital disorder of unknown cause with a population prevalence of 1%-2%. In previous studies we have shown up-regulation of vascular smooth muscle cell (VSMC) apoptosis and cystic medial necrosis in the tunica media of BAV aneurysm tissue similar to Marfan syndrome (MFS) aortic aneurysm. In BAV but not MFS aneurysm we have identified in electron microscopy studies autophagy as an additional cause of VSMC loss. Osteopontin (OPN) exerts pro-inflammatory effects on VSMCs and has recently been shown to stimulate autophagy and down-regulate Notch1. In this study we examined apoptosis, autophagy and expression of OPN in human aortic aneurysm tissue. Thoracic aortic aneurysm tissue was obtained from subjects with BAV (6M; 5F; 31-75yr) and Marfan syndrome (MFS; 5M; 3F; 26-76yr) at the time of surgery. Normal thoracic aorta (TAC; 1M; 4F; 22-56yr) was obtained from organ donors. The number of VSMC..." @default.
- W2942172892 created "2019-05-03" @default.
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- W2942172892 date "2012-11-20" @default.
- W2942172892 modified "2023-09-26" @default.
- W2942172892 title "Abstract 17762: Osteopontin is Associated with Vascular Smooth Muscle Cell Autophagy in Bicuspid Aortic Valve Aneurysm" @default.
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