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- W2945117005 abstract "Necroptosis is a tightly regulated form of necrosis that requires the activation of receptor-interacting protein (RIP) kinases RIPK1 and RIPK3, as well as the RIPK3 substrate mixed lineage kinase domain-like protein (MLKL). Because of membrane rupture, necroptotic cells release damage-associated molecular patterns (DAMPs) that evoke immune responses. Necroptosis is emerging as an important cellular response in the modulation of cancer initiation, progression, and metastasis. Necroptosis of cancer cells is considered to be an immunogenic cell death capable of activating anti-tumor immunity. Necroptosis also participates in the promotion of myeloid cell-induced adaptive immune suppression and thus contributes to oncogenesis. In addition, necroptosis of endothelial cells and tumor cells is conducive to tumor metastasis. In this review, we summarize the current knowledge of the complex role of necroptosis in cancer and discuss the potential of targeting necroptosis components for cancer therapies." @default.
- W2945117005 created "2019-05-29" @default.
- W2945117005 creator A5000246187 @default.
- W2945117005 creator A5003599636 @default.
- W2945117005 creator A5036621543 @default.
- W2945117005 creator A5081534229 @default.
- W2945117005 date "2019-05-01" @default.
- W2945117005 modified "2023-10-17" @default.
- W2945117005 title "Complex roles of necroptosis in cancer" @default.
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- W2945117005 doi "https://doi.org/10.1631/jzus.b1900160" @default.
- W2945117005 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6568224" @default.
- W2945117005 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/31090266" @default.