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- W2945363791 endingPage "16" @default.
- W2945363791 startingPage "1" @default.
- W2945363791 abstract "Reactive oxygen species (ROS) promote carcinogenesis by inducing genetic mutations, activating oncogenes, and raising oxidative stress, which all influence cell proliferation, survival, and apoptosis. Cancer cells display redox imbalance due to increased ROS level compared to normal cells. This unique feature in cancer cells may, therefore, be exploited for targeted therapy. Over the past few decades, natural compounds have attracted attention as potential cancer therapies because of their ability to maintain cellular redox homeostasis with minimal toxicity. Preclinical studies show that bioactive dietary polyphenols exert antitumor effects by inducing ROS-mediated cytotoxicity in cancer cells. These bioactive compounds also regulate cell proliferation, survival, and apoptotic and antiapoptotic signalling pathways. In this review, we discuss (i) how ROS is generated and (ii) regulated and (iii) the cell signalling pathways affected by ROS. We also discuss (iv) the various dietary phytochemicals that have been implicated to have cancer therapeutic effects through their ROS-related functions." @default.
- W2945363791 created "2019-05-29" @default.
- W2945363791 creator A5046221852 @default.
- W2945363791 creator A5048096571 @default.
- W2945363791 creator A5058855547 @default.
- W2945363791 date "2019-05-14" @default.
- W2945363791 modified "2023-10-16" @default.
- W2945363791 title "ROS-Mediated Cancer Cell Killing through Dietary Phytochemicals" @default.
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