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• W2945668830 abstract "Testosterone (T) therapy has garnered widespread public enthusiasm and media attention due to its potential role in age-related T decline in men, commonly known as late-onset hypogonadism (LOH), andropause, or low T syndrome. The serum T concentration gradually declines across the lifespan and the symptoms between aging and hypogonadism overlap. These have led to the speculation that a causal relationship might exists between age-related reduction in serum T concentration and symptoms commonly seen in aging. However, it remains uncertain if T therapy could ameliorate symptoms associated with LOH, without significant risks. Despite the lack of clinical evidence and long term safety data, prescribing rates of T therapy have skyrocketed in many countries (1, 2), leading to efforts by regulatory authorities to limit such inappropriate prescribing practice (3).Importantly, the fundamental question of what constitutes clinically significant LOH was largely unaddressed until recently. Heterogeneity in definitions of LOH and the use of specificity-limited immunoassays for T measurements in many previous epidemiological and interventional studies have precluded robust comparisons across studies (4). Due to the expanding aging population, LOH is becoming an increasingly important topic. We reviewed the evidence from recent population-based studies and intervention trials to provide better understanding of the diagnosis, pathophysiology, and management for LOH." @default.
• W2945668830 created "2019-05-29" @default.
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• W2945668830 date "2019-06-12" @default.
• W2945668830 modified "2023-09-27" @default.
• W2945668830 title "Late-Onset Hypogonadism as Primary Testicular Failure" @default.
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• W2945668830 doi "https://doi.org/10.3389/fendo.2019.00372" @default.
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