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- W2945709947 abstract "All homologues of herpesvirus gH studied to date have been implicated in playing an essential role in viral infection of diverse permissive cell types. However, the role of gH in the mechanism of KSHV infection remains largely unresolved. In this study, we generated a gH-null mutant KSHV and provided evidence that deficiency of gH expression did not affect viral particle assembly or egress. Using the gH-null mutant, we showed that gH was indispensable for KSHV infection of epithelial, endothelial, and fibroblast cells in vitro . This suggests that gH is an important target for the development of a KSHV prophylactic vaccine to prevent initial viral infection." @default.
- W2945709947 created "2019-05-29" @default.
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- W2945709947 date "2019-08-15" @default.
- W2945709947 modified "2023-10-01" @default.
- W2945709947 title "Kaposi Sarcoma-Associated Herpesvirus Glycoprotein H Is Indispensable for Infection of Epithelial, Endothelial, and Fibroblast Cell Types" @default.
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- W2945709947 doi "https://doi.org/10.1128/jvi.00630-19" @default.
- W2945709947 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6675886" @default.
- W2945709947 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/31142670" @default.
- W2945709947 hasPublicationYear "2019" @default.
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