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- W2945747111 abstract "In 20 patients with liver cirrhosis and prolongation of thrombin (Th) and/or thrombin coagulase (ThC) times, fibrin polymerisation was followed at 350 nm after adjustment of fibrinogen concentration. The optical-density values at 10’ (OD 10) were grossly abnormal both with Th and with ThC, but no correlation was found between OD 10 and Th or ThC times. Polymerisation was re-tested after fibrinogen purification with 6 alanine and glycine. ThC time was generally shortened (p < 0.005) although in no case restored to normal. The OD 10 values improved (p < 0.001), but became normal in only a half of the cases, while remaining totally abnormal in 2 cases: PAA gel electrophoresis showed an abnormal band of very low m.w. fibrinogen in 2 out of 10 cases investigated. These results suggest that defective fibrin polymerisation in liver cirrhosis cannot be attributed only to acquired dysfibrinogenaemia. Other features like partially degraded fibrinogen and, especially, still unknown antipolymerizing factor(s)in plasma contribute to the disorder of iibrin formation." @default.
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- W2945747111 date "1979-01-01" @default.
- W2945747111 modified "2023-09-23" @default.
- W2945747111 title "Fibrin Formation in Liver Cirrhosis: Acquired Dysfibrinogenaemia or Hampered Polymerisation?" @default.
- W2945747111 doi "https://doi.org/10.1055/s-0039-1684719" @default.
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