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- W2956691551 abstract "Les moisissures sont un facteur de risque d’asthme, que cela soit chez l’enfant ou l’adulte. Le lien entre exposition, sensibilisation et symptômes n’a pu s’observer nettement que dans l’asthme très sévère à Alternaria chez l’adulte. En revanche, l’exposition aux moisissures dans la petite enfance serait un facteur de risque d’asthme quel que soit le statut atopique ou non atopique. L’exposition ou la sensibilisation aux moisissures prises isolément sont des facteurs de risque d’asthme sévère et/ou d’exacerbations chez l’enfant. Il semblerait même qu’il y ait une relation de cause à effet entre l’exposition et l’asthme. Ce lien semble moins fort chez l’adulte. En revanche, chez l’adulte, la sensibilisation aux moisissures semble déterminer un phénotype d’asthme plus sévère avec un trouble ventilatoire obstructif plus marqué. En effet, les moisissures agissant aussi bien sur l’immunité innée qu’acquise semblent être responsables d’une inflammation Th2 particulièrement marquée conduisant à des asthmes plus sévères. Cependant, à côté des mécanismes immunologiques, des mécanismes toxiques pourraient intervenir. Ainsi, il semble difficile de réduire l’effet des moisissures dans l’apparition de symptômes, notamment respiratoires, au seul mécanisme allergique. Exposure to mould is a potential risk factor for asthma in both adults and children. In adult, the relation between exposure, sensitization and symptoms has been demonstrated in severe asthmatics sensitized to Alternaria. For children, exposure to mould in childhood is a risk factor for asthma in both atopic and non-atopic individuals. Exposure or sensitization to moulds are a risk factor for severe asthma and/or exacerbations in children. There appears to be a causal relationship between exposure and asthma. This link seems less significant in adults. However, in adults mould sensitive asthma seems to determine a phenotype of severe asthma associated with more marked obstructive lung disease. Moulds can stimulate either innate or the acquired immunity. They are responsible for a marked Th2 inflammation leading to more severe asthma. Besides the immunological mechanisms, toxic mechanisms can also intervene. It is therefore not correct to reduce the effect of moulds, particularly in respiratory symptoms, to only allergic mechanisms." @default.
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- W2956691551 date "2019-09-01" @default.
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- W2956691551 title "Pathologies allergiques respiratoires liées aux moisissures de l’habitat" @default.
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- W2956691551 doi "https://doi.org/10.1016/j.rmr.2018.10.619" @default.
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