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• W2958907555 abstract "Aluminum (Al) promotes programmed cell death (PCD) in plants. Although a lot of knowledge about the mechanisms of Al tolerance has been learned, how Al-induced PCD is regulated by nitric oxide (NO) is poorly understood. Mitochondrion is the regulatory center for PCD. We found that Al reduced the level of mitochondrial NO/H2O2, promoted the opening of mitochondrial permeability transition pore, decreased mitochondrial inner membrane potential (∆ψm), and increased caspase-like protease activity. NO-specific scavenger cPTIO enhanced these effects that were reversed by NO donor sodium nitroprusside. Our data suggest that NO suppresses Al-induced PCD by improving mitochondrial physiological properties." @default.
• W2958907555 created "2019-07-23" @default.
• W2958907555 creator A5008048355 @default.
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• W2958907555 date "2019-07-11" @default.
• W2958907555 modified "2023-09-23" @default.
• W2958907555 title "Nitric oxide is a suppressor of aluminum-induced mitochondria and caspase-like protease-dependent programmed cell death in plants" @default.
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• W2958907555 doi "https://doi.org/10.1080/15592324.2019.1640566" @default.
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