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• W2964546133 endingPage "766" @default.
• W2964546133 startingPage "754" @default.
• W2964546133 abstract "CNS injuries, such as traumatic brain injury (TBI), subarachnoid hemorrhage (SAH), intracerebral hemorrhage (ICH), and cerebral ischemic stroke, are important causes of death and long-term disability worldwide. As an important class of pervasive genes involved in many pathophysiological processes, long non-coding RNAs (lncRNAs) have received attention in the past decades. Multiple studies indicate that lncRNAs are abundant in the CNS and have a key role in brain function as well as many neurological disorders, especially in CNS injuries. Several investigations have deciphered that regulation of lncRNAs exert pro-angiogenesis, anti-apoptosis, and anti-inflammation effects in CNS injury via different molecules and pathways, including microRNA (miRNA), nuclear factor kappa-light-chain-enhancer of activated B cells (NF-κB), phosphatidylinositol-4,5-bisphosphate 3-kinase/protein kinase B (PI3K/AKT), Notch, and p53. Thus, lncRNAs show great promise as molecular targets in CNS injuries. In this article, we provide an updated review of the current state of our knowledge about the relationship between lncRNAs and CNS injuries, highlighting the specific roles of lncRNAs in CNS injuries." @default.
• W2964546133 created "2019-08-13" @default.
• W2964546133 creator A5013694779 @default.
• W2964546133 creator A5041100767 @default.
• W2964546133 date "2019-09-01" @default.
• W2964546133 modified "2023-10-14" @default.
• W2964546133 title "Long Non-coding RNA in CNS Injuries: A New Target for Therapeutic Intervention" @default.
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