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- W2964921337 abstract "Background and Aims: Postprandial lipemia (PPL) is associated with atherosclerosis development and cardiovascular diseases. PCSK9 reduces plasma cholesterol by promoting LDLR degradation. We previously showed that PCSK9-deficiency reduced PPL by altering the intestinal chylomicron production and by increasing their hepatic catabolism. Our study aims: i) to assess the relative importance of hepatic or intestinal PCSK9 in PPL regulation ; ii) to assess the efficacy of an anti-PCSK9 monoclonal antibody on PPL in C57BL/6J and LDLR-/- mice ; iii) to determine whether PCSK9-deficiency also reduced PPL in insulinopenic streptozotocin(STZ)-induced diabetic mice." @default.
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- W2964921337 date "2019-08-01" @default.
- W2964921337 modified "2023-09-24" @default.
- W2964921337 title "Pharmacological Inhibition And Hepatic-Deficiency Of Pcsk9 Reduce Post-Prandial Lipemia In Mice" @default.
- W2964921337 doi "https://doi.org/10.1016/j.atherosclerosis.2019.06.008" @default.
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