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- W2969794868 endingPage "241" @default.
- W2969794868 startingPage "223" @default.
- W2969794868 abstract "There are growing genetic, transcriptomic and proteomic data pointing to the complexity of Alzheimer's disease (AD) pathogenesis. Unbiased “omics” approaches are essential for the future development of effective AD research, which will need to be combined and personalized, given that multiple distinct pathways can drive AD pathology. It is essential to gain a better understanding of the AD pathogenesis subtype variety and to develop several distinct therapeutic approaches tailored to address this diversity, as well as the common presence of mixed pathologies. These nonmutually exclusive therapeutic approaches include the targeting of multiple toxic oligomeric species concurrently, targeting the apolipoprotein E/amyloid β interaction and the modulation of innate immunity, as well as more “out of the box” ideas such as targeting infectious agents that may play a role in AD." @default.
- W2969794868 created "2019-08-29" @default.
- W2969794868 creator A5060656369 @default.
- W2969794868 creator A5063931891 @default.
- W2969794868 date "2019-01-01" @default.
- W2969794868 modified "2023-09-25" @default.
- W2969794868 title "Future horizons in Alzheimer's disease research" @default.
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