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• W2970522219 abstract "The genomes of positive-sense RNA viruses have an intrinsic capacity to serve directly as mRNAs upon viral entry into a host cell. These RNAs often lack a 5′ cap structure and 3′ polyadenylation sequence, requiring unconventional strategies for cap-independent translation and subversion of the cellular RNA degradation machinery. For tombusviruses, critical translational regulatory elements are encoded within the 3′ untranslated region of the viral genomes. Here we describe RNA modifications occurring within the genomes of Panicum mosaic virus (PMV), a prototypical tombusvirus, and its satellite agents (i.e., satellite virus and noncoding satellite RNAs), all of which depend on the PMV-encoded RNA polymerase for replication. The atypical RNAs are defined by terminal polyadenylation and truncation events within the 3′ untranslated region of the PMV genome. These modifications are reminiscent of host-mediated RNA degradation strategies and likely represent a previously underappreciated defense mechanism against invasive nucleic acids." @default.
• W2970522219 created "2019-09-05" @default.
• W2970522219 creator A5001501679 @default.
• W2970522219 creator A5028719348 @default.
• W2970522219 creator A5031435050 @default.
• W2970522219 date "2019-08-27" @default.
• W2970522219 modified "2023-10-14" @default.
• W2970522219 title "<i>Panicum Mosaic Virus</i> and Its Satellites Acquire RNA Modifications Associated with Host-Mediated Antiviral Degradation" @default.
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• W2970522219 doi "https://doi.org/10.1128/mbio.01900-19" @default.
• W2970522219 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6712398" @default.
• W2970522219 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/31455653" @default.
• W2970522219 hasPublicationYear "2019" @default.
• W2970522219 type Work @default.
• W2970522219 sameAs 2970522219 @default.
• W2970522219 citedByCount "7" @default.
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