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• W2971448696 abstract "Aim: To investigate whether and how CD4 + T cells contribute to ILC2 activation during respiratory syncytial virus (RSV) infection. Methods: The methods of flow cytometry, quantitative PCR and ELISA were used in the present study. Results: Depletion of CD4 + T cells diminished the numbers of lung ILC2s as well as their ability to produce type 2 cytokines. CD4 + T cell-mediated ILC2 activation is related to IL-2. The main cellular source of IL-2 was CD4 + T cells. Depletion of CD4 + T cells decreased IL-2 levels in the lungs of RSV-infected mice. IL-2 can directly stimulate ILC2 proliferation and promote ILC2s to produce cytokines. Treatment of mice with neutralizing anti-IL-2 monoclonal antibodies diminished ILC2 activation. Conclusion: These results suggest that CD4 + T cells contribute to RSV-induced ILC2 activation partly via producing IL-2." @default.
• W2971448696 created "2019-09-12" @default.
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• W2971448696 date "2019-10-01" @default.
• W2971448696 modified "2023-10-18" @default.
• W2971448696 title "Essential role of CD4⁺ T cells for the activation of group 2 innate lymphoid cells during respiratory syncytial virus infection in mice" @default.
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• W2971448696 doi "https://doi.org/10.2217/imt-2019-0084" @default.
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