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- W2974819782 abstract "Abstract Pulmonary arterial hypertension (PAH) is a vasculopathy of the lungs characterized by cellular and structural changes that increase vascular resistance, causing an increase in right-ventricular afterload that can lead to ventricular hypertrophy and remodeling, and heart failure. The prognosis of PAH is very poor and the underlying causes remain untreatable. Since PAH is a disease involving the heart and pulmonary vasculature and multiple scales of biological organization, a quantitative understanding of PAH requires multiphysics, multiscale mathematical models that integrate from the molecular to cellular, tissue, organ, and system scales. In this review, we highlight the most recent modeling efforts by scale and discuss new models that will be needed to develop a fully integrated multiscale and multiphysics analysis of the heart and lungs in PAH." @default.
- W2974819782 created "2019-09-26" @default.
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- W2974819782 date "2019-09-01" @default.
- W2974819782 modified "2023-09-24" @default.
- W2974819782 title "Multiscale modeling of ventricular–vascular dysfunction in pulmonary arterial hypertension" @default.
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- W2974819782 doi "https://doi.org/10.1016/j.cobme.2019.09.004" @default.
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