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- W29771412 abstract "William Gibson might have included cancer and inflammation as an example of “apophonia”. The perception of this connection, however, has been anything but spontaneous. Cancer is generally thought of as disordered cell growth driven by the accumulation of genes that confer a growth advantage or prevent cellular senescence [1]. Inflammation is thought of primarily as a non-immune, acute host-defense process. When persistent, chronic inflammation and the associated immune response together play a role in several diseases including rheumatoid arthritis, asthma, and inflammatory bowel disease. The thoughtful clinician and scientist reflecting on the extraordinary advances in cancer biology and the search for causes of cancer are disappointed with how our premises have not yet translated into more effective therapy. Those studying diseases associated with inflammation raise a similar concern, with many of the therapeutic targets seemingly intractable. What are we missing? The editors of this volume have addressed the relatedness of these seemingly “unrelated” fields to reveal “meaningfulness” that is now more perceptible. This revelation could provide a synergistic base to allow the generation of new questions and to accelerate innovation in understanding and treatment of these diseases." @default.
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- W29771412 date "2004-01-01" @default.
- W29771412 modified "2023-09-25" @default.
- W29771412 title "Inflammation, necrosis, and cancer" @default.
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- W29771412 doi "https://doi.org/10.1007/978-3-0348-7861-6_9" @default.
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