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- W2977979435 abstract "Background: Acute esophageal necrosis (AEN), also known as ‘black esophagus,’ is a rare clinical entity characterized by endoscopic findings of circumferential black-appearing distal esophageal mucosa. While the etiology of AEN is unclear, it is thought to be due to a combination of ischemic insult, corrosive injury, and decreased function of mucosal barrier systems. Here we report a case of a middle-aged man with AEN who was seen at our University Hospital in the past year. Methods: In the past year, our University Hospital managed one patient who was found to have AEN. We review the history, presentation, imaging, endoscopic techniques used for diagnosis and management, and treatment for this patient case. Results: A 59-year-old man with poorly controlled type II diabetes presented with hyperglycemic hyperosmolar state (HHS) and coffee ground emesis. Ten days prior, he was admitted to an outside institution for obstructive jaundice and was found to have a distal common bile duct (CBD) stricture. He had an internal-external percutaneous biliary drain placed after a failed Endoscopic Retrograde Cholangiopancreatography (ERCP). On this admission, computed tomography of the abdomen and pelvis revealed a multi-lobar pneumonia, right abdominal percutaneous drainage catheter terminating in the gallbladder fossa, and marked gastric distention to level of gastroduodenal junction (Fig. 1). Once blood glucose levels were stabilized, he underwent an esophagogastroduodenoscopy (EGD) that revealed diffuse black mucosal abnormalities throughout the esophagus terminating at the gastroesophageal junction (Fig. 2). These findings were consistent with AEN. His esophageal findings were not a contraindication to endoscopy and successful rendezvous ERCP was then performed, revealing a high-grade distal CBD stricture due to acute-on-chronic pancreatitis. A biliary sphincterotomy was performed with complete removal of the percutaneous biliary drain and placement of a plastic biliary stent. The patient was maintained on his insulin regimen and was started on high dose proton pump inhibitor (PPI) therapy with resolution of coffee ground emesis. Upon follow up four months later, repeat EGD demonstrated complete mucosal healing (Fig. 3).Figure 1Figure 2Figure 3Discussion: Poorly controlled diabetes and HHS serve as risk factors for the development of AEN. Optimal treatment of AEN includes management of predisposing risk factors and use of high dose proton pump inhibitors." @default.
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- W2977979435 date "2016-10-01" @default.
- W2977979435 modified "2023-10-02" @default.
- W2977979435 title "Black Esophagus: Acute Esophageal Necrosis in Hyperglycemic Hyperosmolar State" @default.
- W2977979435 doi "https://doi.org/10.14309/00000434-201610001-01663" @default.
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