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- W2977982255 abstract "Purpose: Patient was referred to our Service for assesment of ascites and hepatosplenomegaly. He was complaining of abdominal dullness, nausea and fatigue over the past 4 weeks. In July 2009, he was diagnosed with septic arthritis, brucellosis and he was performed arthroscopic debridement and given brucella treatment. In control examination pulmonary thromboembolism was diagnosed, his medication was warfarin but he discontinued it. 4 weeks later he referred to our service. Computed tomography revealed hepatosplenomegaly, suspected thrombus formation in the vena cava inferior at the suprarenal and infrarenal levels, heterogenity of liver parenchyma, massive ascites (Fig. 1). Doppler ultrasonography revealed very slow flow signal, hepatic venous collaterals. Endoscopy revealed Grade I-II esophagus varices. Inferior/superior vena cavagraphy showed total occlusion of inferior vena cava and lower extremity venous drainage was flowing directly to superior vena cava via paravertebral collaterals and hemiazygous vein (Fig. 2). We have reported a very rare alliance of multiple hypercoagulable situations (decreased activitiy, decreased antigenic concentration of AT, low protein C activity, heterozygote Factor V Leiden mutation) which caused Budd-Chiari Syndrom. BCS is a rare but crucial situation and thrombofilia is a basis for BCS, and a complete search for thrombofilia is very critical.FigureFigure" @default.
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- W2977982255 date "2011-10-01" @default.
- W2977982255 modified "2023-09-25" @default.
- W2977982255 title "A Hepatic Outflow Obstruction (Budd-Chiari Syndrome) Case due to Antithrombin III Deficiency" @default.
- W2977982255 doi "https://doi.org/10.14309/00000434-201110002-00764" @default.
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