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- W2979702978 abstract "Host responses are often ineffective at clearing Mycoplasma bovis infection and may contribute to the pathogenesis of disease. M bovis possesses a surprisingly large repertoire of strategies to evade and modulate host responses. Unopsonized M bovis impairs phagocytosis and killing by neutrophils and macrophages. Apoptosis of neutrophils and lymphocytes is enhanced, whereas it is delayed in macrophages. Both proinflammatory and antiinflammatory cytokines are stimulated during M bovis infection depending on the cell type and location, and overall systemic responses tend to have a T-helper 2 bias. M bovis reduces proliferation of T cells and, in chronic infection, causes T-cell exhaustion." @default.
- W2979702978 created "2019-10-18" @default.
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- W2979702978 date "2019-11-01" @default.
- W2979702978 modified "2023-10-02" @default.
- W2979702978 title "Mycoplasma bovis" @default.
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- W2979702978 doi "https://doi.org/10.1016/j.cvfa.2019.08.003" @default.
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