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- W2980014643 abstract "I have recently reiterated that the cross-bridge is a calcium ATPase that is inhibited by magnesium and this arises because in normal hearts Myosin binding Protein-C prevents the use of MgATP as rate limiting substrate ensuring that Ca2+ replaces Mg2+ in the excitation pathway. Here I revisit the studies on [Ca2+] dependency of ATPase and tension under diastolic stretch with a different conclusion on Hill coefficients. This reveals the underlying mechanisms of the Frank-Starling Law and Hypertrophic myopathy are not the same, the former being kinase controlled." @default.
- W2980014643 created "2019-10-18" @default.
- W2980014643 creator A5047532769 @default.
- W2980014643 date "2019-10-07" @default.
- W2980014643 modified "2023-10-17" @default.
- W2980014643 title "The Mechanisms of the Frank-Starling Law and Familial Cardiomyopathy are Different. The Function of Myosin Binding Protein-C is Retained on Myocyte Length Increase and Force Generated is Kinase controlled" @default.
- W2980014643 doi "https://doi.org/10.20944/preprints201910.0068.v1" @default.
- W2980014643 hasPublicationYear "2019" @default.
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