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- W2980877881 abstract "A fundamental question regarding the etiology of amyotrophic lateral sclerosis (ALS) is whether the various gene mutations associated with the disease converge on a single molecular pathway or act through multiple pathways to trigger neurodegeneration. Notably, several of the genes and cellular processes implicated in ALS have also been linked to frontotemporal dementia (FTD), suggesting these two diseases share common origins with varied clinical presentations. Scientists are rapidly identifying ALS/FTD suppressors that act on conserved pathways from invertebrates to vertebrates to alleviate degeneration. The elucidation of such genetic modifiers provides insight into the molecular pathways underlying this rapidly progressing neurodegenerative disease, while also revealing new targets for therapeutic development." @default.
- W2980877881 created "2019-10-25" @default.
- W2980877881 creator A5006662673 @default.
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- W2980877881 creator A5077883174 @default.
- W2980877881 date "2019-10-16" @default.
- W2980877881 modified "2023-10-18" @default.
- W2980877881 title "Phenotypic Suppression of ALS/FTD-Associated Neurodegeneration Highlights Mechanisms of Dysfunction" @default.
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