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• W2981314588 abstract "Existing animal models do not replicate all aspects of abdominal aortic aneurysms (AAAs), including the rupture mechanisms. From histopathological analyses conducted in humans, it has been found that the vasa vasorum of the AAA wall is the starting point of circulatory failure and that bulging and dilatation of the abdominal aorta occurs through inflammation and tissue degeneration. We created a new animal model (the hypoperfusion-induced model) of AAAs. In this study, we describe the current animal models of AAAs and present the utility of our new model of AAAs. Existing animal models do not replicate all aspects of abdominal aortic aneurysms (AAAs), including the rupture mechanisms. From histopathological analyses conducted in humans, it has been found that the vasa vasorum of the AAA wall is the starting point of circulatory failure and that bulging and dilatation of the abdominal aorta occurs through inflammation and tissue degeneration. We created a new animal model (the hypoperfusion-induced model) of AAAs. In this study, we describe the current animal models of AAAs and present the utility of our new model of AAAs." @default.
• W2981314588 created "2019-10-25" @default.
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• W2981314588 date "2020-02-01" @default.
• W2981314588 modified "2023-10-06" @default.
• W2981314588 title "The Role of Animal Models in Elucidating the Etiology and Pathology of Abdominal Aortic Aneurysms: Development of a Novel Rupture Mechanism Model" @default.
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• W2981314588 doi "https://doi.org/10.1016/j.avsg.2019.08.082" @default.
• W2981314588 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/31626940" @default.
• W2981314588 hasPublicationYear "2020" @default.
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