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• W2981844080 abstract "Recognition of non-self structures on donor cells represents the main immunological barrier in solid organ transplantation. The human leukocyte antigens (HLA) are considered the most important non-self (allo)antigens in transplantation. Long-term graft attrition is mainly caused by the formation of alloreactive antibodies that are directed against non-self structures (i.e., epitopes) on cell surface proteins. Recently published data provided evidence for a similar importance of non-HLA mismatches between donors and recipients in acute rejection as well as long-term kidney allograft survival. These data suggest a broader concept of immunological non-self that goes beyond HLA incompatibility and expands the current concept of polymorphic non-self epitopes on cell surface molecules from HLA to non-HLA targets. Amino acid substitutions caused by single nucleotide variants in protein-coding genes or complete loss of gene expression represent the basis for polymorphic residues in both HLA and non-HLA molecules. To better understand these novel insights in non-HLA alloimmunity, we will first review basic principles of the alloimmune response with a focus on the HLA epitope concept in donor-specific antibody formation before discussing key publications on non-HLA antibodies." @default.
• W2981844080 created "2019-11-01" @default.
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• W2981844080 date "2019-11-28" @default.
• W2981844080 modified "2023-10-17" @default.
• W2981844080 title "Novel insights into non‐HLA alloimmunity in kidney transplantation" @default.
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• W2981844080 doi "https://doi.org/10.1111/tri.13546" @default.
• W2981844080 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6972536" @default.
• W2981844080 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/31650645" @default.

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