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- W2983453772 abstract "Herpesviruses constitute a large family of disease-causing DNA viruses. Each herpesvirus strain is capable of infecting particular organisms with a specific cell tropism. Upon infection, pattern recognition receptors (PRRs) recognize conserved viral features to trigger signaling cascades that culminate in the production of interferons and pro-inflammatory cytokines. To invoke a proper immune response while avoiding collateral tissue damage, signaling proteins involved in these cascades are tightly regulated by post-translational modifications (PTMs). Herpesviruses have developed strategies to subvert innate immune signaling pathways in order to ensure efficient viral replication and achieve persistent infection. The ability of these viruses to control the proteins involved in these signaling cascades post-translationally, either directly via virus-encoded enzymes or indirectly through the deregulation of cellular enzymes, has been widely reported. This ability provides herpesviruses with a powerful tool to shut off or restrict host antiviral and inflammatory responses. In this review, we highlight recent findings on the herpesvirus-mediated post-translational control along PRR-mediated signaling pathways." @default.
- W2983453772 created "2019-11-22" @default.
- W2983453772 creator A5007865277 @default.
- W2983453772 creator A5025300076 @default.
- W2983453772 creator A5025988358 @default.
- W2983453772 creator A5032174342 @default.
- W2983453772 creator A5036130322 @default.
- W2983453772 creator A5084208079 @default.
- W2983453772 date "2019-11-14" @default.
- W2983453772 modified "2023-10-16" @default.
- W2983453772 title "Post-translational Control of Innate Immune Signaling Pathways by Herpesviruses" @default.
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