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• W2983613740 abstract "IntroductionDengue infection causes significant morbidity and mortality in over 125 countries worldwide, and its incidence is on the rise. Currently, no therapy is available beyond supportive care. In fact, corticosteroids are used therapeutically for a broad spectrum of diseases including autoimmune, allergic, inflammatory diseases and organ transplant rejection. However, only a few studies were done to evaluate the effectiveness of corticosteroids in dengue infection, although the immune pathology of dengue is similar to other diseases treated effectively by corticosteroids for several decades.ObjectivesThis review is aimed at identifying biological actions of steroids at molecular and receptor level in dengue immune pathology after reviewing pharmacological and immunological research findings of corticosteroids and dengue.MethodsWe searched medline/pubmed and Google scholar for publications with the search terms ‘dengue’ and ‘steroid’, ‘corticosteroid’, ‘prednisolone’, ‘methylprednisolone’ or ‘dexamethasone’ in the title and abstract. Then the publications were analyzed according the action of steroids in dengue pathology under different subheadings.ResultsThe results are presented under four categories and it shows that corticosteroids can suppress cells involved in innate immunity, T cells, B cells and antibodies,complements and heamatological manifestations in dengue pathology.ConclusionThis article explains strong supportive evidence for actions of corticosteroids in dengue pathology at receptors and molecular levels. Therefore it is suggested that a gold standard steroid protocol for each phase of dengue pathology that can be tested further with a double blind control trial study." @default.
• W2983613740 created "2019-11-22" @default.
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• W2983613740 date "2020-06-01" @default.
• W2983613740 modified "2023-09-27" @default.
• W2983613740 title "Corticosteroid actions on dengue immune pathology; A review article" @default.
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• W2983613740 doi "https://doi.org/10.1016/j.cegh.2019.11.001" @default.
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